Transient hyperinsulinism in asphyxiated newborn infants.

K. Schultz, G. Soltész

Research output: Contribution to journalArticle

10 Citations (Scopus)

Abstract

Hypoglycemia in birth asphyxiated infants is attributed to glycogen depletion. We observed three term AGA (Appropriate for Gestational Age) infants with birth asphyxia, who developed hyperinsulinemic hypoglycemia postnatally. All had inappropriately high serum insulin concentrations for their blood glucose levels, and needed glucose infusion rates of greater than 8 mg/kg/min for several days to maintain normoglycemia. All infants recovered spontaneously.

Original languageEnglish
Pages (from-to)47-52
Number of pages6
JournalActa paediatrica Hungarica
Volume31
Issue number1
Publication statusPublished - 1991

ASJC Scopus subject areas

  • Pediatrics, Perinatology, and Child Health

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