The appearance of circulating anti-heart antibodies and myocardium-bound antibodies was demonstrated after experimental myocardial infarction induced by coronary ligation or high doses of isoproterenol in rats. Rat heart homogenate and mitochondria at 500 μ/ml protein level inhibited the migration of leucocytes obtained from rats following myocardial infarction. The immunological consequences were similar in infarctions induced by coronary ligation or isoproterenol treatment.
|Number of pages||5|
|Journal||Acta physiologica Academiae Scientiarum Hungaricae|
|Publication status||Published - Dec 1 1976|
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