Abstract
Insulin treatment following subtotal hepatectomy caused a long lasting change in the binding capacity of hepatic insulin receptors. The insulin binding increased in females and decreased in males in the insulin treated animals (during regeneration) fourteen days after the operation. The tendency of changes was very similar to those which had been caused by neonatal insulin treatment.
Original language | English |
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Pages (from-to) | 461-464 |
Number of pages | 4 |
Journal | Acta Physiologica Hungarica |
Volume | 73 |
Issue number | 4 |
Publication status | Published - 1989 |
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ASJC Scopus subject areas
- Physiology
Cite this
Hormonal imprinting in adults : insulin exposure during regeneration alters the later binding capacity of the hepatic insulin receptors. / Csaba, G.; Inczefi-Gonda, A.; Dobozy, O.
In: Acta Physiologica Hungarica, Vol. 73, No. 4, 1989, p. 461-464.Research output: Contribution to journal › Article
}
TY - JOUR
T1 - Hormonal imprinting in adults
T2 - insulin exposure during regeneration alters the later binding capacity of the hepatic insulin receptors.
AU - Csaba, G.
AU - Inczefi-Gonda, A.
AU - Dobozy, O.
PY - 1989
Y1 - 1989
N2 - Insulin treatment following subtotal hepatectomy caused a long lasting change in the binding capacity of hepatic insulin receptors. The insulin binding increased in females and decreased in males in the insulin treated animals (during regeneration) fourteen days after the operation. The tendency of changes was very similar to those which had been caused by neonatal insulin treatment.
AB - Insulin treatment following subtotal hepatectomy caused a long lasting change in the binding capacity of hepatic insulin receptors. The insulin binding increased in females and decreased in males in the insulin treated animals (during regeneration) fourteen days after the operation. The tendency of changes was very similar to those which had been caused by neonatal insulin treatment.
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UR - http://www.scopus.com/inward/citedby.url?scp=0024824079&partnerID=8YFLogxK
M3 - Article
C2 - 2686351
AN - SCOPUS:0024824079
VL - 73
SP - 461
EP - 464
JO - Physiology International
JF - Physiology International
SN - 2498-602X
IS - 4
ER -