Clinical and pathologic features of lineage 2 West Nile virus infections in birds of prey in Hungary

K. Erdélyi, K. Ursu, E. Ferenczi, L. Szeredi, Ferenc Rátz, József Skáre, T. Bakonyi

Research output: Contribution to journalArticle

69 Citations (Scopus)

Abstract

In the southeast of Hungary a sparrow hawk (Accipiter nisus) and several goshawk (Accipiter gentilis) fledglings succumbed to encephalitis manifesting as an acute neurological disease during the summers of 2004 and 2005. Both years the causative agent was identified as a lineage 2 West Nile virus. This is the first description of clinical, pathological and immunohistochemical findings of infection caused by a neuroinvasive, lineage 2 West Nile virus and the first evidence of its circulation in continental Europe.

Original languageEnglish
Pages (from-to)181-188
Number of pages8
JournalVector-Borne and Zoonotic Diseases
Volume7
Issue number2
DOIs
Publication statusPublished - Jun 2007

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Raptors
West Nile virus
Hungary
Virus Diseases
Hawks
Sparrows
Acute Disease
Encephalitis
Infection

Keywords

  • Arbovirus(es)
  • Epidemiology
  • West Nile
  • Zoonotic

ASJC Scopus subject areas

  • Infectious Diseases
  • Parasitology

Cite this

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abstract = "In the southeast of Hungary a sparrow hawk (Accipiter nisus) and several goshawk (Accipiter gentilis) fledglings succumbed to encephalitis manifesting as an acute neurological disease during the summers of 2004 and 2005. Both years the causative agent was identified as a lineage 2 West Nile virus. This is the first description of clinical, pathological and immunohistochemical findings of infection caused by a neuroinvasive, lineage 2 West Nile virus and the first evidence of its circulation in continental Europe.",
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AU - Erdélyi, K.

AU - Ursu, K.

AU - Ferenczi, E.

AU - Szeredi, L.

AU - Rátz, Ferenc

AU - Skáre, József

AU - Bakonyi, T.

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AB - In the southeast of Hungary a sparrow hawk (Accipiter nisus) and several goshawk (Accipiter gentilis) fledglings succumbed to encephalitis manifesting as an acute neurological disease during the summers of 2004 and 2005. Both years the causative agent was identified as a lineage 2 West Nile virus. This is the first description of clinical, pathological and immunohistochemical findings of infection caused by a neuroinvasive, lineage 2 West Nile virus and the first evidence of its circulation in continental Europe.

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